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A cross-sectional study of determinants of indoor environmental exposures in households with and without chronic exposure to biomass fuel smoke 2014 Division of Pulmonary and Critical Care, School of Medicine, Johns Hopkins University, 1800 Orleans Ave, Suite 9121, Baltimore, MD, USA. wcheckl1@jhmi.edu.
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Environmental health : a global access science source
Periodical, Abbrev.
Environ.Health
Pub Date Free Form
24-Mar
Volume
13
Issue
1
Start Page
21
Other Pages
069X-13-21
Notes
LR: 20150514; GR: 5R25TW009340/TW/FIC NIH HHS/United States; GR: P30ES003819/ES/NIEHS NIH HHS/United States; GR: R00HL096955/HL/NHLBI NIH HHS/United States; JID: 101147645; 0 (Particulate Matter); 0 (Smoke); 7U1EE4V452 (Carbon Monoxide); OID: NLM: PMC3978
Place of Publication
England
ISSN/ISBN
1476-069X; 1476-069X
Accession Number
PMID: 24655424
Language
eng
SubFile
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; IM
DOI
10.1186/1476-069X-13-21 [doi]
Output Language
Unknown(0)
PMID
24655424
Abstract
BACKGROUND: Burning biomass fuels indoors for cooking is associated with high concentrations of particulate matter (PM) and carbon monoxide (CO). More efficient biomass-burning stoves and chimneys for ventilation have been proposed as solutions to reduce indoor pollution. We sought to quantify indoor PM and CO exposures in urban and rural households and determine factors associated with higher exposures. A secondary objective was to identify chronic vs. acute changes in cardiopulmonary biomarkers associated with exposure to biomass smoke. METHODS: We conducted a census survey followed by a cross-sectional study of indoor environmental exposures and cardiopulmonary biomarkers in the main household cook in Puno, Peru. We measured 24-hour indoor PM and CO concentrations in 86 households. We also measured PM2.5 and PM10 concentrations gravimetrically for 24 hours in urban households and during cook times in rural households, and generated a calibration equation using PM2.5 measurements. RESULTS: In a census of 4903 households, 93% vs. 16% of rural vs. urban households used an open-fire stove; 22% of rural households had a homemade chimney; and
Descriptors
Links
Book Title
Database
Publisher
Data Source
Authors
Pollard,S.L., Williams,D.L., Breysse,P.N., Baron,P.A., Grajeda,L.M., Gilman,R.H., Miranda,J.J., Checkley,W., CRONICAS Cohort Study Group
Original/Translated Title
URL
Date of Electronic
20140324
PMCID
PMC3978088
Editors
Low correlation between household carbon monoxide and particulate matter concentrations from biomass-related pollution in three resource-poor settings 2015 Division of Pulmonary and Critical Care, School of Medicine, Johns Hopkins University, Baltimore, USA.; Division of Pulmonary and Critical Care, School of Medicine, Johns Hopkins University, Baltimore, USA.; Division of Pulmonary and Critical Care, School
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Environmental research
Periodical, Abbrev.
Environ.Res.
Pub Date Free Form
Oct
Volume
142
Issue
Start Page
424
Other Pages
431
Notes
LR: 20160708; CI: Published by Elsevier Inc.; GR: HHSN268200900033C/HL/NHLBI NIH HHS/United States; GR: HHSN268200900033C/PHS HHS/United States; JID: 0147621; 0 (Particulate Matter); 7U1EE4V452 (Carbon Monoxide); NIHMS712528; OID: NLM: NIHMS712528 [Availa
Place of Publication
United States
ISSN/ISBN
1096-0953; 0013-9351
Accession Number
PMID: 26245367
Language
eng
SubFile
Journal Article; Research Support, N.I.H., Extramural; IM
DOI
10.1016/j.envres.2015.07.012 [doi]
Output Language
Unknown(0)
PMID
26245367
Abstract
Household air pollution from the burning of biomass fuels is recognized as the third greatest contributor to the global burden of disease. Incomplete combustion of biomass fuels releases a complex mixture of carbon monoxide (CO), particulate matter (PM) and other toxins into the household environment. Some investigators have used indoor CO concentrations as a reliable surrogate of indoor PM concentrations; however, the assumption that indoor CO concentration is a reasonable proxy of indoor PM concentration has been a subject of controversy. We sought to describe the relationship between indoor PM2.5 and CO concentrations in 128 households across three resource-poor settings in Peru, Nepal, and Kenya. We simultaneously collected minute-to-minute PM2.5 and CO concentrations within a meter of the open-fire stove for approximately 24h using the EasyLog-USB-CO data logger (Lascar Electronics, Erie, PA) and the personal DataRAM-1000AN (Thermo Fisher Scientific Inc., Waltham, MA), respectively. We also collected information regarding household construction characteristics, and cooking practices of the primary cook. Average 24h indoor PM2.5 and CO concentrations ranged between 615 and 1440 mug/m(3), and between 9.1 and 35.1 ppm, respectively. Minute-to-minute indoor PM2.5 concentrations were in a safe range (
Descriptors
Links
Book Title
Database
Publisher
Data Source
Authors
Klasen,E.M., Wills,B., Naithani,N., Gilman,R.H., Tielsch,J.M., Chiang,M., Khatry,S., Breysse,P.N., Menya,D., Apaka,C., Carter,E.J., Sherman,C.B., Miranda,J.J., Checkley,W., COCINAS Trial Working Group
Original/Translated Title
URL
Date of Electronic
20150731
PMCID
PMC4932836
Editors
Time trends of polycyclic aromatic hydrocarbon exposure in New York City from 2001 to 2012: assessed by repeat air and urine samples 2014 Division of Pulmonary, Allergy and Critical Care of Medicine, Department of Medicine, College of Physicians and Surgeons, Columbia University, PH8E-101, 630W. 168 Street, New York, NY 10032, United States.; Division of Pulmonary, Allergy and Critical Care
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Environmental research
Periodical, Abbrev.
Environ.Res.
Pub Date Free Form
May
Volume
131
Issue
Start Page
95
Other Pages
103
Notes
LR: 20150806; CI: Copyright (c) 2014; GR: P01 ES009600/ES/NIEHS NIH HHS/United States; GR: P01ES09600/ES/NIEHS NIH HHS/United States; GR: P30 ES009089/ES/NIEHS NIH HHS/United States; GR: P30ES09089/ES/NIEHS NIH HHS/United States; GR: P50 ES015905/ES/NIEHS
Place of Publication
United States
ISSN/ISBN
1096-0953; 0013-9351
Accession Number
PMID: 24709094
Language
eng
SubFile
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; IM
DOI
10.1016/j.envres.2014.02.017 [doi]
Output Language
Unknown(0)
PMID
24709094
Abstract
BACKGROUND: Exposure to air pollutants including polycyclic aromatic hydrocarbons (PAH), and specifically pyrene from combustion of fuel oil, coal, traffic and indoor sources, has been associated with adverse respiratory health outcomes. However, time trends of airborne PAH and metabolite levels detected via repeat measures over time have not yet been characterized. We hypothesized that PAH levels, measured repeatedly from residential indoor and outdoor monitors, and childrens urinary concentrations of PAH metabolites, would decrease following policy interventions to reduce traffic-related air pollution. METHODS: Indoor PAH (particle- and gas-phase) were collected for two weeks prenatally (n=98), at age 5/6 years (n=397) and age 9/10 years (n=198) since 2001 and at all three age-points (n=27). Other traffic-related air pollutants (black carbon and PM2.5) were monitored indoors simultaneous with PAH monitoring at ages 5/6 (n=403) and 9/10 (n=257) between 2005 and 2012. One third of the homes were selected across seasons for outdoor PAH, BC and PM2.5 sampling. Using the same sampling method, ambient PAH, BC and PM2.5 also were monitored every two weeks at a central site between 2007 and 2012. PAH were analyzed as semivolatile PAH (e.g., pyrene; MW 178-206) ( summation operator8PAH(semivolatile): Including pyrene (PYR), phenanthrene (PHEN), 1-methylphenanthrene (1-MEPH), 2-methylphenanthrene (2-MEPH), 3-methylphenanthrene (3-MEPH), 9-methylphenanthrene (9-MEPH), 1,7-dimethylphenanthrene (1,7-DMEPH), and 3,6-dimethylphenanthrene (3,6-DMEPH)) and the sum of eight nonvolatile PAH ( summation operator8PAH(nonvolatile): Including benzo[a]anthracene (BaA), chrysene/iso-chrysene (Chry), benzo[b]fluoranthene (BbFA), benzo[k]fluoranthene (BkFA), benzo[a]pyrene (BaP), indeno[1,2,3-c,d]pyrene (IP), dibenzo[a,h]anthracene (DahA), and benzo[g,h,i]perylene (BghiP); MW 228-278). A spot urine sample was collected from children at child ages 3, 5, 7 and 9 between 2001 and 2012 and analyzed for 10 PAH metabolites. RESULTS: Modest declines were detected in indoor BC and PM2.5 levels between 2005 and 2012 (Annual percent change [APC]=-2.08% [p=0.010] and -2.18% [p=0.059] for BC and PM2.5, respectively), while a trend of increasing pyrene levels was observed in indoor and outdoor samples, and at the central site during the comparable time periods (APC=4.81%, 3.77% and 7.90%, respectively; p
Descriptors
Links
Book Title
Database
Publisher
Elsevier Inc
Data Source
Authors
Jung,K.H., Liu,B., Lovinsky-Desir,S., Yan,B., Camann,D., Sjodin,A., Li,Z., Perera,F., Kinney,P., Chillrud,S., Miller,R.L.
Original/Translated Title
URL
Date of Electronic
20140405
PMCID
PMC4031101
Editors
Cigarette smoking and the risk of Barrett's esophagus 2009 Division of Research, Kaiser Permanente, 2000 Broadway, Oakland, CA 94612, USA. ai.kubo@kp.org
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Cancer causes & control : CCC
Periodical, Abbrev.
Cancer Causes Control
Pub Date Free Form
Apr
Volume
20
Issue
3
Start Page
303
Other Pages
311
Notes
LR: 20151119; GR: F32 DK081271/DK/NIDDK NIH HHS/United States; GR: K08 DK 002697/DK/NIDDK NIH HHS/United States; GR: K08 DK002697/DK/NIDDK NIH HHS/United States; GR: K08 DK002697-05/DK/NIDDK NIH HHS/United States; GR: R01 DK 63616/DK/NIDDK NIH HHS/United
Place of Publication
Netherlands
ISSN/ISBN
1573-7225; 0957-5243
Accession Number
PMID: 18853262
Language
eng
SubFile
Journal Article; Research Support, N.I.H., Extramural; IM
DOI
10.1007/s10552-008-9244-4 [doi]
Output Language
Unknown(0)
PMID
18853262
Abstract
INTRODUCTION: We examined the association between smoking and the risk of Barrett's esophagus (BE), a metaplastic precursor to esophageal adenocarcinoma. METHODS: We conducted a case-control study within the Kaiser Permanente Northern California population. Patients with a new diagnosis of BE (n = 320) were matched to persons with gastroesophageal reflux disease (GERD) (n = 316) and to population controls (n = 317). Information was collected using validated questionnaires from direct in-person interviews and electronic databases. Analyses used multivariate unconditional logistic regression that controlled for age, gender, race, and education. RESULTS: Ever smoking status, smoking intensity (pack-years), and smoking cessation were not associated with the risk of BE. Stratified analyses suggested that ever smoking may be associated with an increased risk of BE among some groups (compared to population controls): persons with long-segment Barrett's esophagus (odds ratio [OR] = 1.72, 95% confidence interval [CI] 1.12-2.63); subjects without GERD symptoms (OR = 3.98, 95% CI 1.58-10.0); obese subjects (OR = 3.38, 95% CI 1.46-7.82); and persons with a large abdominal circumference (OR = 3.02, 95% CI (1.18-2.75)). CONCLUSION: Smoking was not a strong or consistent risk factor for BE in a large community-based study, although associations may be present in some population subgroups.
Descriptors
Adenocarcinoma/epidemiology, Adolescent, Adult, Aged, Alcohol Drinking/adverse effects, Barrett Esophagus/epidemiology, Body Mass Index, California/epidemiology, Case-Control Studies, Confidence Intervals, Confounding Factors (Epidemiology), Databases, Factual, Esophageal Neoplasms/epidemiology, Gastroesophageal Reflux/epidemiology, Geography, Humans, Incidence, Interviews as Topic, Logistic Models, Middle Aged, Obesity/complications, Odds Ratio, Precancerous Conditions/complications, Reproducibility of Results, Risk Factors, Smoking/adverse effects, Surveys and Questionnaires, Young Adult
Links
Book Title
Database
Publisher
Data Source
Authors
Kubo,A., Levin,T. R., Block,G., Rumore,G., Quesenberry,C. P.,Jr, Buffler,P., Corley,D. A.
Original/Translated Title
URL
Date of Electronic
20081014
PMCID
PMC2649684
Editors
Abdominal obesity and body mass index as risk factors for Barrett&#39;s esophagus 2007 Division of Research, Kaiser Permanente, Oakland, California 94612, USA. douglas.corley@kp.org <douglas.corley@kp.org>
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Gastroenterology
Periodical, Abbrev.
Gastroenterology
Pub Date Free Form
Jul
Volume
133
Issue
1
Start Page
34
Other Pages
41; quiz 311
Notes
LR: 20080102; GR: K08 DK02697/DK/NIDDK NIH HHS/United States; GR: R01 DK63616/DK/NIDDK NIH HHS/United States; JID: 0374630; CIN: Gastroenterology. 2007 Jul;133(1):343-5. PMID: 17631153; CIN: Gastroenterology. 2007 Dec;133(6):2075; author reply 2075-6. PMI
Place of Publication
United States
ISSN/ISBN
0016-5085; 0016-5085
Accession Number
PMID: 17631128
Language
eng
SubFile
Journal Article; Research Support, N.I.H., Extramural; AIM; IM
DOI
S0016-5085(07)00832-3 [pii]
Output Language
Unknown(0)
PMID
17631128
Abstract
BACKGROUND: Barrett's esophagus is a strong risk factor for esophageal adenocarcinoma, but little is known about its associations with body mass index (BMI) or abdominal obesity. METHODS: We conducted a case-control study within the Kaiser Permanente Northern California population. Persons with a new diagnosis of Barrett's esophagus (cases) were matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's esophagus and to population controls. Subjects completed questionnaires and an anthropometric examination. RESULTS: We interviewed 320 cases, 316 patients with GERD, and 317 controls. There was a general association between Barrett's esophagus and a larger abdominal circumference (independent of BMI) compared with population controls (odds ratio, 2.24; 95% confidence interval, 1.21-4.15; circumference, >80 cm vs 80 cm and no significant trend for further increases in circumference. There was a trend for association compared with patients with GERD (test for trend, P = .03). There was no association between Barrett's esophagus and BMI. Abdominal circumference was associated with GERD symptom severity (odds ratio, 1.86; 95% confidence interval, 1.03-3.38; risk of severe weekly GERD, per 10-cm circumference); adjustment for GERD partially attenuated the association between Barrett's esophagus and circumference. CONCLUSIONS: Waist circumference, but not BMI, had some modest independent associations with the risk of Barrett's esophagus. The findings provide partial support for the hypothesis that abdominal obesity contributes to GERD, which may in turn increase the risk of Barrett's esophagus.
Descriptors
Abdominal Fat, Adult, Aged, Barrett Esophagus/epidemiology, Body Mass Index, California/epidemiology, Case-Control Studies, Education, Medical, Continuing, Female, Gastroesophageal Reflux/epidemiology, Humans, Incidence, Male, Middle Aged, Obesity/epidemiology/pathology, Risk Factors, Thigh, Waist-Hip Ratio
Links
Book Title
Database
Publisher
Data Source
Authors
Corley,D. A., Kubo,A., Levin,T. R., Block,G., Habel,L., Zhao,W., Leighton,P., Quesenberry,C., Rumore,G. J., Buffler,P. A.
Original/Translated Title
URL
Date of Electronic
20070425
PMCID
Editors
Alcohol types and sociodemographic characteristics as risk factors for Barrett&#39;s esophagus 2009 Division of Research, Kaiser Permanente, Oakland, California, USA.
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Gastroenterology
Periodical, Abbrev.
Gastroenterology
Pub Date Free Form
Mar
Volume
136
Issue
3
Start Page
806
Other Pages
815
Notes
LR: 20140914; GR: F32 DK081271/DK/NIDDK NIH HHS/United States; GR: K08DK002697/DK/NIDDK NIH HHS/United States; GR: R01 DK063616/DK/NIDDK NIH HHS/United States; GR: R01 DK063616-05/DK/NIDDK NIH HHS/United States; GR: R01 DK63616/DK/NIDDK NIH HHS/United Sta
Place of Publication
United States
ISSN/ISBN
1528-0012; 0016-5085
Accession Number
PMID: 19111726
Language
eng
SubFile
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; AIM; IM
DOI
10.1053/j.gastro.2008.11.042 [doi]
Output Language
Unknown(0)
PMID
19111726
Abstract
BACKGROUND & AIMS: Little is known about the effects of alcohol use and sociodemographics on the risk of Barrett's esophagus, a precursor to esophageal adenocarcinoma. We evaluated the association between alcohol use, alcohol type, sociodemographic profiles, other lifestyle factors, and the risk of Barrett's esophagus. METHODS: With the use of a case-control study within the Kaiser Permanente Northern California membership, patients with a new diagnosis of Barrett's esophagus (n = 320) diagnosed between 2002 and 2005 were matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to population controls (n = 317). We collected information using validated questionnaires during direct in-person interviews. Analyses used multivariate unconditional logistic regression. RESULTS: Total alcohol use was not significantly associated with the risk of Barrett's esophagus, although stratification by beverage type showed an inverse association for wine drinkers compared with nondrinkers (>/=7 drinks of wine per week vs none: odds ratio, 0.44; 95% confidence interval, 0.20-0.99; multivariate analysis). Among population controls, those who preferred wine were more likely to have college degrees and regularly take vitamin supplements than those who preferred beer or liquor, although adjustment for these factors or GERD symptoms did not eliminate the inverse association between wine consumption and Barrett's esophagus. Education status was significantly inversely associated with the risk of Barrett's esophagus. CONCLUSIONS: There are associations between alcohol types, socioeconomic status, and the risk of Barrett's esophagus. Although choice of alcoholic beverages was associated with several factors, multiple adjustments (including for GERD) did not eliminate the association between alcohol and Barrett's esophagus. Further research to evaluate the associations among socioeconomic status, GERD, and Barrett's esophagus is warranted.
Descriptors
Adult, Aged, Alcohol Drinking/epidemiology, Barrett Esophagus/epidemiology, Beer/statistics & numerical data, California/epidemiology, Case-Control Studies, Female, Gastroesophageal Reflux/epidemiology, Humans, Life Style, Male, Middle Aged, Risk Factors, Socioeconomic Factors, Wine/statistics & numerical data, Young Adult
Links
Book Title
Database
Publisher
Data Source
Authors
Kubo,A., Levin,T. R., Block,G., Rumore,G. J., Quesenberry,C. P.,Jr, Buffler,P., Corley,D. A.
Original/Translated Title
URL
Date of Electronic
20081127
PMCID
PMC2675884
Editors
Adiponectin May Modify the Risk of Barrett's Esophagus in Patients With Gastroesophageal Reflux Disease 2015 Division of Research, Kaiser Permanente, Oakland, California.; Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, California; Department of Nutrition, University of California, Davis, California.; Departme
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
Periodical, Abbrev.
Clin.Gastroenterol.Hepatol.
Pub Date Free Form
Dec
Volume
13
Issue
13
Start Page
2256
Other Pages
64.e1-3
Notes
LR: 20151123; CI: Copyright (c) 2015; GR: R01 DK063616/DK/NIDDK NIH HHS/United States; GR: R01 DK63616/DK/NIDDK NIH HHS/United States; GR: R56 DK087748/DK/NIDDK NIH HHS/United States; GR: R56 DK087748/DK/NIDDK NIH HHS/United States; JID: 101160775; 0 (Adi
Place of Publication
United States
ISSN/ISBN
1542-7714; 1542-3565
Accession Number
PMID: 25632808
Language
eng
SubFile
Journal Article; Research Support, N.I.H., Extramural; IM
DOI
10.1016/j.cgh.2015.01.009 [doi]
Output Language
Unknown(0)
PMID
25632808
Abstract
BACKGROUND & AIMS: Abdominal obesity and increasing body mass index are risk factors for esophageal adenocarcinoma and its main precursor, Barrett's esophagus; however, there are no known biological mechanisms for these associations or regarding why only some patients with gastroesophageal reflux disease develop Barrett's esophagus. We evaluated the association between Barrett's esophagus and multimers of an adipose-associated hormone, adiponectin. METHODS: We conducted a case-control study evaluating the associations between adiponectin (total, high-molecular-weight, and low-/medium-molecular-weight) and Barrett's esophagus within the Kaiser Permanente Northern California population. Patients with a new diagnosis of Barrett's esophagus (cases) were matched to patients with gastroesophageal reflux disease (GERD) without Barrett's esophagus and to population controls. RESULTS: Complete serologic and epidemiologic data were available for 284 cases, 294 GERD controls, and 285 population controls. Increasing adiponectin levels were a risk factor for Barrett's esophagus among patients with GERD (total adiponectin fourth vs first quartile odds ratio [OR], 1.96; 95% confidence interval [CI], 1.17-3.27; high-molecular-weight adiponectin OR, 1.65; 95% CI, 1.00-2.73; low-/medium-molecular-weight adiponectin OR, 2.18; 95% CI, 1.33-3.56), but not compared with population controls. The associations were significantly stronger among patients reporting frequent GERD symptoms and among smokers (P values interaction
Descriptors
Links
Book Title
Database
Publisher
AGA Institute. Published by Elsevier Inc
Data Source
Authors
Almers,L.M., Graham,J.E., Havel,P.J., Corley,D.A.
Original/Translated Title
URL
Date of Electronic
20150126
PMCID
PMC4515407
Editors
An in vitro investigation of a comparison of bond strengths of composite to etched and air-abraded human enamel surfaces 2006 Division of Restorative Dentistry, Bristol Dental School, Lower Maudlin Street, Bristol BS1 2LY, UK.
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Journal of prosthodontics : official journal of the American College of Prosthodontists
Periodical, Abbrev.
J.Prosthodont.
Pub Date Free Form
Jan-Feb
Volume
15
Issue
1
Start Page
2
Other Pages
8
Notes
LR: 20100826; CI: Copyright (c) 2006; JID: 9301275; 0 (Composite Resins); 0 (Polymethacrylic Acids); 0 (Prime and Bond NT); 0 (Resin Cements); 0 (TPH spectrum); ppublish
Place of Publication
United States
ISSN/ISBN
1059-941X; 1059-941X
Accession Number
PMID: 16433645
Language
eng
SubFile
Comparative Study; Journal Article; D
DOI
JOPR062 [pii]
Output Language
Unknown(0)
PMID
16433645
Abstract
PURPOSE: The purposes of the study were to measure the tensile bond strength of composite resin to human enamel specimens that had been either etched or air-abraded, and to compare the quality of the marginal seal, through the assessment of microleakage, of composite resin to human enamel specimens that had been either etched or air-abraded. MATERIALS AND METHODS: Thirty mandibular molar teeth were decoronated and sectioned mesio-distally to produce six groups, each containing ten specimens that were embedded in acrylic resin using a jig. In each of the four treatment groups, the specimen surfaces were treated by either abrasion with 27 or 50 microm alumina at 4 mm or 20 mm distance, and a composite resin was bonded to the treated surfaces in a standardized manner. In the two control groups the specimens were treated with 15 seconds exposure to 36% phosphoric acid gel and then similarly treated before being stored in sterile water for 1 week. All specimens were then subjected to tensile bond strength testing at either 1 or 5 mm/min crosshead speed. For the microleakage study, the degree of dye penetration was measured 32 times for each treatment group, using a neutral methylene blue dye at the interface between composite and either 27 or 50 microm air-abraded tooth structure or etched enamel surfaces. RESULTS: The mean bond strength values recorded for Group 1 (phosphoric acid etch, 5 mm/min crosshead speed) was 25.4 MPa; Group 2 (phosphoric acid etch, 1 mm/min), 22.2 MPa; Group 3 (27 microm alumina at 4 mm distance), 16.8 MPa; Group 4 (50 microm alumina at 4 mm distance), 16.9 MPa; Group 5 (27 microm alumina at 20 mm distance), 4.2 MPa; and for Group 6 (50 microm alumina at 20 mm distance) 3.4 MPa. An analysis of variance (ANOVA) demonstrated significant differences among the groups, and a multiple comparison test (Tukey) demonstrated that conventionally etched specimens had a greater bond strength than air-abraded specimen groups. No significant difference in dye penetration could be demonstrated among the groups (p= 0.58). CONCLUSIONS: Composite resin applied to enamel surfaces prepared using an acid etch procedure exhibited higher bond strengths than those prepared with air abrasion technology. The abrasion particle size did not affect the bond strength produced, but the latter was adversely affected by the distance of the air abrasion nozzle from the enamel surface. The crosshead speed of the bond testing apparatus had no effect on the bond strengths recorded. The marginal seal of composite to prepared enamel was unaffected by the method of enamel preparation.
Descriptors
Acid Etching, Dental, Air Abrasion, Dental, Analysis of Variance, Composite Resins, Dental Bonding/methods, Dental Enamel, Dental Leakage/prevention & control, Dental Stress Analysis, Humans, Mandible, Molar, Polymethacrylic Acids, Resin Cements, Statistics, Nonparametric, Surface Properties, Tensile Strength, Tooth Preparation/methods
Links
Book Title
Database
Publisher
by The American College of Prosthodontists
Data Source
Authors
Gray,G. B., Carey,G. P., Jagger,D. C.
Original/Translated Title
URL
Date of Electronic
PMCID
Editors
The periodontal status of irregular dental attenders 1994 Division of Restorative Dentistry, School of Clinical Dentistry, Queens University, Belfast, Northern Ireland.
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Journal of clinical periodontology
Periodical, Abbrev.
J.Clin.Periodontol.
Pub Date Free Form
Sep
Volume
21
Issue
8
Start Page
544
Other Pages
548
Notes
LR: 20061115; JID: 0425123; ppublish
Place of Publication
DENMARK
ISSN/ISBN
0303-6979; 0303-6979
Accession Number
PMID: 7989618
Language
eng
SubFile
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; D; IM
DOI
Output Language
Unknown(0)
PMID
7989618
Abstract
The aim of this investigation was to assess the prevalence and severity of periodontal destruction in irregular dental attenders. 50 subjects aged between 20 and 49 years completed a questionnaire and had a periodontal examination. Measurements of plaque, calculus, bleeding, probing depth and clinical attachment level were made at 4 proximal sites per tooth. Results were compared to those for 132 regular attenders. The irregular attenders had more sites with plaque (46.3 +/- 27% versus 17.3 +/- 21.1%) than the regulars and significantly more sites which bled (53.7 +/- 20.4% versus 33.6 +/- 20.7%) and with calculus (35 +/- 21.1% versus 13.4 +/- 12.8%). However, the irregulars had virtually the same number of teeth as the regular attenders, 25.6 +/- 3.3 compared with 25.4 +/- 4.3. Irregular attenders had slightly deeper mean probing depths (2.9 +/- 0.4 versus 2.7 +/- 0.4 mm) but the prevalence and extent of attachment loss was no different between the groups. It was concluded that the prevalence and severity of destructive periodontal disease as indicated by periodontal attachment loss was not related to the regularity of dental attendance in the groups studied.
Descriptors
Adult, Dental Calculus/etiology/pathology, Dental Care, Dental Plaque/etiology/pathology, Female, Furcation Defects/etiology, Gingival Hemorrhage/etiology/pathology, Humans, Male, Middle Aged, Oral Hygiene, Periodontal Attachment Loss/etiology/pathology, Periodontal Diseases/etiology/pathology, Periodontal Pocket/etiology/pathology, Prevalence, Risk Factors, Smoking, Social Class, Tooth/pathology
Links
Book Title
Database
Publisher
Data Source
Authors
Mullally,B. H., Linden,G. J.
Original/Translated Title
URL
Date of Electronic
PMCID
Editors
Predictive global trends in the incidence and mortality of pancreatic cancer based on geographic location, socio-economic status, and demographic shift 2016 Division of Surgical Oncology, Department of Surgery, University of Nebraska Medical Center, Omaha, Nebraska.; Section of Gastroenterology, Department of Medicine, Boston University Medical Center, Boston, Massachusetts.; Department of Surgery, Gundersen
Source Type
Print(0)
Ref Type
Journal Article
Periodical, Full
Journal of surgical oncology
Periodical, Abbrev.
J.Surg.Oncol.
Pub Date Free Form
11-Aug
Volume
Issue
Start Page
Other Pages
Notes
LR: 20160811; CI: (c) 2016; JID: 0222643; OTO: NOTNLM; 2016/01/27 [received]; 2016/07/25 [accepted]; aheadofprint
Place of Publication
ISSN/ISBN
1096-9098; 0022-4790
Accession Number
PMID: 27511902
Language
ENG
SubFile
JOURNAL ARTICLE
DOI
10.1002/jso.24410 [doi]
Output Language
Unknown(0)
PMID
27511902
Abstract
BACKGROUND AND OBJECTIVES: Pancreatic Cancer (PC) is a lethal malignancy that accounts for about 4% of cancer-related deaths worldwide. The aim of this study is to describe the influence of geography (based on WHO regions), socio-economic development (based on Human Development Index [HDI]) and demographic shift on the temporal trends in global incidence and mortality of PC. METHODS: Data (2012-2030) relating to the incidence, mortality of PC and demographic shifts based on WHO regions and HDI areas were extracted from GLOBOCAN 2012. Linear regression was used to evaluate trends in total incidence and mortality. RESULTS: We noted a definite association between PC and higher socio-economic status. Advanced age (age >/=65) contributed to the rising burden in all socio-economic regions of the world except in the Low Human Development (LHD) countries where the disease predominantly affected population
Descriptors
Links
Book Title
Database
Publisher
Wiley Periodicals, Inc
Data Source
Authors
Are,C., Chowdhury,S., Ahmad,H., Ravipati,A., Song,T., Shrikandhe,S., Smith,L.
Original/Translated Title
URL
Date of Electronic
20160811
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